Semax is a synthetic peptide derived from a fragment of adrenocorticotropic hormone (ACTH) that lacks hormonal activity but retains neuroactive properties. It is widely studied in neuroscience and neuropharmacology research for its influence on neurotrophic factors and synaptic signaling.
In research settings, semax is examined for its effects on brain-derived neurotrophic factor (BDNF) expression, neurotransmitter modulation, and neural plasticity pathways. It has also been explored in experimental models focused on cognitive processing, stress-response signaling, and cerebral blood flow regulation.
Research involving semax centers on neuroprotective signaling, cognitive-related pathways, and central nervous system modulation rather than therapeutic or clinical application.
